Journal of Molecular Signaling
Published by Ubiquity Press
eISSN : 1750-2187
Abbreviation : J. Mol. Signal.
Aims & Scope
Journal of Molecular Signaling encompasses all the molecular aspects of cell signaling involving receptors, G-proteins, kinases, phosphatases, and transcription factors in regulating cell proliferation, differentiation, apoptosis, and oncogenesis in mammalian cells.
Molecular signaling is an exponentially growing field that encompasses different molecular aspects of cell signaling underlying normal and pathological conditions.
Specifically, the journal focuses on the normal or aberrant molecular mechanisms involving receptors, G-proteins, kinases, phosphatases, and transcription factors in regulating cell proliferation, differentiation, apoptosis, and oncogenesis in mammalian cells.
This also includes the genetic and epigenetic changes that modulate the signaling properties of cells and the resultant physiological conditions.
View Aims & ScopeMetrics & Ranking
SJR (SCImago Journal Rank)
| Year | Value |
|---|---|
| 2024 | 0.255 |
Quartile
| Year | Value |
|---|---|
| 2024 | Q4 |
h-index
| Year | Value |
|---|---|
| 2024 | 36 |
Journal Rank
| Year | Value |
|---|---|
| 2024 | 18406 |
Journal Citation Indicator
| Year | Value |
|---|---|
| 2024 | 3 |
Abstracting & Indexing
Journal is indexed in leading academic databases, ensuring global visibility and accessibility of our peer-reviewed research.
Subjects & Keywords
Journal’s research areas, covering key disciplines and specialized sub-topics in Biochemistry, Genetics and Molecular Biology, designed to support cutting-edge academic discovery.
Licensing & Copyright
This journal operates under an Open Access model. Articles are freely accessible to the public immediately upon publication. The content is licensed under a Creative Commons Attribution 4.0 International License (CC BY 4.0), allowing users to share and adapt the work with proper attribution.
Copyright remains with the author(s), and no permission is required for non-commercial use, provided the original source is cited.