Aging Cell

Aims & Scope

Aging Cell welcome submissions in the following areas: -Genetics and functional genomics: mutations affecting longevity; gene homologies; organismal and cellular aging; gene manipulation. -Signaling and gene expression: mechanisms linking age-associated changes with phenotypes and physiology; intracellular signaling; interactions between cells and tissues; hormonal, immune, and inflammatory systems. -Cell proliferation, aging and death: replicative senescence; apoptosis; telomere biology and other intrinsic and extrinsic influences; chronological cellular aging; phenotypes of aging cells. -Cell stress and damage: extrinsic and intrinsic influences of free radicals on cells and tissues; free radical defense and damage; free radicals as signaling molecules; stress and aging. -Stem cells and aging: effects of age on stem cell generation; migration and homeostasis; stem cell-niche interactions; regulatory mechanisms. -Integrative physiology: outcomes of aging processes at organismal, cellular and molecular levels. -Biodemography and comparative studies: population and cross-species comparative studies. -New theories of aging and longevity: discussion at the broadest level of established and novel theories of aging and longevity.

Metrics & Ranking

Abstracting & Indexing

Journal is indexed in leading academic databases, ensuring global visibility and accessibility of our peer-reviewed research.

• Scopus
• Google Scholar
• Directory of Open Access Journals (DOAJ)
• Web of Science
• Medline
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• Embase

Subjects & Keywords

Journal’s research areas, covering key disciplines and specialized sub-topics in Biochemistry, Genetics and Molecular Biology, designed to support cutting-edge academic discovery.

Licensing & Copyright

This journal operates under an Open Access model. Articles are freely accessible to the public immediately upon publication. The content is licensed under a Creative Commons Attribution 4.0 International License (CC BY 4.0), allowing users to share and adapt the work with proper attribution.

Copyright remains with the author(s), and no permission is required for non-commercial use, provided the original source is cited.

Policy Links

This section provides access to essential policy documents, guidelines, and resources related to the journal’s publication and submission processes.

• Aims scope
• Homepage
• Oa statement
• Author instructions
• License terms
• Review url
• Board url
• Copyright url
• Plagiarism url
• Preservation url
• Apc url
• License

Plagiarism Policy

This journal follows a plagiarism policy. All submitted manuscripts are screened using reliable plagiarism detection software to ensure originality and academic integrity. Authors are responsible for proper citation and acknowledgment of all sources, and any form of plagiarism, including self-plagiarism, will not be tolerated.

For more details, please refer to our official: Plagiarism Policy.

APC Details

The journal’s Article Processing Charge (APC) policies support open access publishing in Biochemistry, Genetics and Molecular Biology, ensuring accessibility and quality in research dissemination.

This journal requires an Article Processing Charge (APC) to support open access publishing, covering peer review, editing, and distribution. The current APC is 3,350.00 USD. Learn more.

Explore journals without APCs for alternative publishing options.

Most Cited Articles

The Most Cited Articles section features the journal's most impactful research, based on citation counts. These articles have been referenced frequently by other researchers, indicating their significant contribution to their respective fields.

• The Achilles’ heel of senescent cells: from transcriptome to senolytic drugs

  Citation: 1980

  Authors: Yi, Tamara, Tamar, Adam C., Husheng, Nino, Allyson K., Yuji, Gene B., Marc, Steven P., Nicholas F., Jordan D., Carolyn M., Grace C., Nathan K., Jonathan D., Joshua N., Sundeep, Michael B., Sara J., Heike, Aditi U., Jing, Debora, Akaitz, Yuan Yuan, Amira S., Diana C., Tokio, Paul D., Laura J., James L.

• Senescence‐associated β‐galactosidase is lysosomal β‐galactosidase

  Citation: 1153

  Authors: Bo Yun, Jung A., Jun Sub, Amelia, Kimberly, Edward C., Wim J., Daniel, Eun Seong

• Identification of a novel senolytic agent, navitoclax, targeting the Bcl‐2 family of anti‐apoptotic factors

  Citation: 930

  Authors: Yi, Tamara, Heike, Haiming M., Yuanyuan Y., Michael B., Tamar, Nino, Kurt O., Cory B., Jonathan D., Laura J., Paul D., James L.

• Fat tissue, aging, and cellular senescence

  Citation: 888

  Authors: Tamara, Dean E., Thomas, Jan, Joseph, Heidi, Sundeep, Michael D., James L.

• <scp>DNA</scp> methylation and healthy human aging

  Citation: 767

  Authors: Meaghan J., Sarah J., Michael S.

• Lifespan extension by conditions that inhibit translation in <i>Caenorhabditis elegans</i>

  Citation: 761

  Authors: Malene, Stefan, Douglas, Nataliya, Seung‐Jae, Cynthia

• Aging activates adipogenic and suppresses osteogenic programs in mesenchymal marrow stroma/stem cells: the role of PPAR‐γ2 transcription factor and TGF‐β/BMP signaling pathways

  Citation: 690

  Authors: Elena J., Kui, David A., Beata

• Uncoupled and surviving: individual mice with high metabolism have greater mitochondrial uncoupling and live longer

  Citation: 659

  Authors: John R., Darren A., Colin, Sam, Jane S., Paula, Ela, Diane M., Maria S., Martin D.

• Methionine‐deficient diet extends mouse lifespan, slows immune and lens aging, alters glucose, T4, IGF‐I and insulin levels, and increases hepatocyte MIF levels and stress resistance

  Citation: 646

  Authors: Richard A., Gretchen, Yayi, James M., Robert, Michael

• Age‐related intrinsic changes in human bone‐marrow‐derived mesenchymal stem cells and their differentiation to osteoblasts

  Citation: 633

  Authors: Shuanhu, Joel S., Michael W., Julie P., Carolyn, Meryl S., Julie