Redox Biology

Aims & Scope

Redox Biology is a forum for novel research, methods and review articles in redox biology in the areas of both health and disease.

Acceptable paper types are research articles (short or full communications), methods, mini-reviews, and commentaries in the following areas: -Nutrition- Polyphenolics- Cancer- Metabolism- Cardiovascular- Diabetes- Inflammation- Aging- Neuroscience- Cell and Molecular Biology- Cell Signaling- Bioenergetic.

Redox Biology will also consider research articles focused in chemical or biochemical mechanisms of redox biology, if these include data demonstrating effects in physiologically relevant models.

Studies of uncharacterized complex mixtures of natural products are not a suitable area of focus for the journal.

Studies using commercial, undefined kits as the sole or primary assay for redox related changes which are not validated using other methods will not be considered for publication.

Redox Biology introduces Graphical Redox Reviews.

These reviews will summarize schematically key concepts, established and novel ideas in redox biology which will be accessible to a broad audience and freely downloaded as PowerPoint slides for use in teaching or conference presentations.

Metrics & Ranking

Abstracting & Indexing

Journal is indexed in leading academic databases, ensuring global visibility and accessibility of our peer-reviewed research.

• Scopus
• Google Scholar
• Directory of Open Access Journals (DOAJ)
• Web of Science
• Embase

Subjects & Keywords

Journal’s research areas, covering key disciplines and specialized sub-topics in Biochemistry, Genetics and Molecular Biology and Chemistry, designed to support cutting-edge academic discovery.

Licensing & Copyright

This journal operates under an Open Access model. Articles are freely accessible to the public immediately upon publication. The content is licensed under a Creative Commons Attribution 4.0 International License (CC BY 4.0), allowing users to share and adapt the work with proper attribution.

Copyright remains with the author(s), and no permission is required for non-commercial use, provided the original source is cited.

Policy Links

This section provides access to essential policy documents, guidelines, and resources related to the journal’s publication and submission processes.

• Aims scope
• Homepage
• Oa statement
• Author instructions
• License terms
• Review url
• Board url
• Copyright url
• Plagiarism url
• Preservation url
• Apc url
• License

Plagiarism Policy

This journal follows a plagiarism policy. All submitted manuscripts are screened using reliable plagiarism detection software to ensure originality and academic integrity. Authors are responsible for proper citation and acknowledgment of all sources, and any form of plagiarism, including self-plagiarism, will not be tolerated.

For more details, please refer to our official: Plagiarism Policy.

APC Details

The journal’s Article Processing Charge (APC) policies support open access publishing in Biochemistry, Genetics and Molecular Biology and Chemistry, ensuring accessibility and quality in research dissemination.

This journal requires an Article Processing Charge (APC) to support open access publishing, covering peer review, editing, and distribution. The current APC is 3,560.00 EUR. Learn more.

Explore journals without APCs for alternative publishing options.

Most Cited Articles

The Most Cited Articles section features the journal's most impactful research, based on citation counts. These articles have been referenced frequently by other researchers, indicating their significant contribution to their respective fields.

• Oxidative stress: a concept in redox biology and medicine

  Citation: 2294

  Authors: Helmut

• Oxidative stress and the amyloid beta peptide in Alzheimer’s disease

  Citation: 1812

  Authors: C., M., D., P., C., F.

• NRF2 plays a critical role in mitigating lipid peroxidation and ferroptosis

  Citation: 1783

  Authors: Matthew, Raul, Donna D.

• Hydrogen peroxide as a central redox signaling molecule in physiological oxidative stress: Oxidative eustress

  Citation: 1731

  Authors: Helmut

• ROS and the DNA damage response in cancer

  Citation: 1653

  Authors: Upadhyayula Sai, Bryce W.Q., Balamurugan A., Anand D.

• Calcium and ROS: A mutual interplay

  Citation: 1281

  Authors: Agnes, Katharina, Sona, Olga

• Reperfusion injury and reactive oxygen species: The evolution of a concept

  Citation: 1195

  Authors: D. Neil, Peter R.

• The Keap1-Nrf2 pathway: Mechanisms of activation and dysregulation in cancer

  Citation: 1122

  Authors: Emilia, Suvi M., Hanna, Anna-Liisa

• Mitochondrial electron transport chain: Oxidative phosphorylation, oxidant production, and methods of measurement

  Citation: 1047

  Authors: Deirdre, Andrea, Sruti

• Role of advanced glycation end products in cellular signaling

  Citation: 966

  Authors: Christiane, Kathleen, Elisa, Anne, Tilman, Andreas