NeuroSignals

Aims & Scope

Neurosignals is an international journal dedicated to publishing original articles and reviews in the field of neuronal communication.

Novel findings related to signaling molecules, channels and transporters, pathways and networks that are associated with development and function of the nervous system are welcome.

The scope of the journal includes genetics, molecular biology, bioinformatics, (patho)physiology, (patho)biochemistry, pharmacology & toxicology, imaging and clinical neurology & psychiatry.

Reported observations should significantly advance our understanding of neuronal signaling in health & disease and be presented in a format applicable to an interdisciplinary readership.

Metrics & Ranking

Abstracting & Indexing

Journal is indexed in leading academic databases, ensuring global visibility and accessibility of our peer-reviewed research.

• Scopus
• Google Scholar
• Directory of Open Access Journals (DOAJ)
• Embase

Subjects & Keywords

Journal’s research areas, covering key disciplines and specialized sub-topics in Neuroscience, designed to support cutting-edge academic discovery.

Licensing & Copyright

This journal operates under an Open Access model. Articles are freely accessible to the public immediately upon publication. The content is licensed under a Creative Commons Attribution 4.0 International License (CC BY 4.0), allowing users to share and adapt the work with proper attribution.

Copyright remains with the author(s), and no permission is required for non-commercial use, provided the original source is cited.

Policy Links

This section provides access to essential policy documents, guidelines, and resources related to the journal’s publication and submission processes.

• Aims scope
• Homepage
• Oa statement
• Author instructions
• License terms
• Review url
• Board url
• Copyright url
• Apc url
• License

APC Details

The journal’s Article Processing Charge (APC) policies support open access publishing in Neuroscience, ensuring accessibility and quality in research dissemination.

This journal does not charge a mandatory Article Processing Charge (APC). However, optional open access publication may incur fees based on the publisher’s policies.

Explore journals without APCs for alternative publishing options.

Most Cited Articles

The Most Cited Articles section features the journal's most impactful research, based on citation counts. These articles have been referenced frequently by other researchers, indicating their significant contribution to their respective fields.

• Significance of Melatonin in Antioxidative Defense System: Reactions and Products

  Citation: 438

  Authors: Dun-Xian, Lucien C., Russel J., Wen-Bo, Malgorzata, Juan R.

• Resveratrol-Activated AMPK/SIRT1/Autophagy in Cellular Models of Parkinson’s Disease

  Citation: 409

  Authors: Yuncheng, Xinqun, Julie Xiaohong, Wenjie, Weidong, Zhen, Joseph, Tianhong

• Physiological Roles for G Protein-Regulated Adenylyl Cyclase Isoforms: Insights from Knockout and Overexpression Studies

  Citation: 319

  Authors: Rachna, Carmen W.

• The Role of Mitogen-Activated Protein Kinase Pathways in Alzheimer’s Disease

  Citation: 316

  Authors: Xiongwei, Hyoung-gon, Arun K., George, Mark A.

• Multifunctional Activities of Green Tea Catechins in Neuroprotection

  Citation: 285

  Authors: Silvia A., Yael, Lydia, Hailin, Orly, Tamar, Moussa B.H.

• FKBP Family Proteins: Immunophilins with Versatile Biological Functions

  Citation: 274

  Authors: Cong Bao, Ye, Sirano, Ho Sup

• The Origin of the Molecular Diversity and Functional Anchoring of Cholinesterases

  Citation: 217

  Authors: Jean

• Brain-Derived Neurotrophic Factor Promotes Central Nervous System Myelination via a Direct Effect upon Oligodendrocytes

  Citation: 213

  Authors: Junhua, Agnes W., Melanie M., Maarten, Trevor J., Simon S.

• Search for Natural Products Related to Regeneration of the Neuronal Network

  Citation: 209

  Authors: Chihiro, Tomoharu, Katsuko

• Neuroprotective Role of Vascular Endothelial Growth Factor: Signalling Mechanisms, Biological Function, and Therapeutic Potential

  Citation: 207

  Authors: Ian