Molecular Pharmacology

Aims & Scope

Molecular Pharmacology welcomes original reports of technically robust and scientifically sound research providing new insights into molecular mechanisms relevant to pharmacology, pharmaceutical sciences, and drug discovery across therapeutic modalities.

Examples include studies of drug-receptor interactions, the impact of receptor function on cell or system physiology, receptor-linked intra- or intercellular signaling systems, enzyme or transporter function, drug delivery, drug metabolism, pharmacogenomics, ion channel function, transcriptional mechanisms, molecular mechanisms underlying disease, mechanisms of agonism, antagonism, allosteric modulation, and signaling bias.

Work focused on the major receptor types (GPCRs, receptor tyrosine kinases, nuclear receptors, etc.) and ion channels, for example, are encouraged.

Manuscripts that identify novel drug targets, approaches to treat disease or protect tissues from damage, mechanisms of action for xenobiotics, or insights into toxicologic mechanisms are also welcome, as are studies that report the development of novel reagents or technologies that are of broad applicability or yield new molecular insights.

Reports that include translational studies, relevant to any of the molecular topics mentioned above, are also encouraged.

Studies involving molecular or mathematical modeling relevant to drug design or drug action will also be considered if they include new experimental data or test existing experimental data to support the model(s).

The journal welcomes minireview articles addressing timely issues in the field of molecular pharmacology.

View Aims & Scope

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Metrics & Ranking

Impact Factor

Year	Value
2025	3
2024	3.20

SJR (SCImago Journal Rank)

Year	Value
2024	1.052

Quartile

Year	Value
2024	Q1

h-index

Year	Value
2024	215

Journal Rank

Year	Value
2024	4444

Journal Citation Indicator

Year	Value
2024	574

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Abstracting & Indexing

Journal is indexed in leading academic databases, ensuring global visibility and accessibility of our peer-reviewed research.

• Scopus
• Google Scholar
• Web of Science
• Embase

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Subjects & Keywords

Journal’s research areas, covering key disciplines and specialized sub-topics in Biochemistry, Genetics and Molecular Biology and Pharmacology, Toxicology and Pharmaceutics, designed to support cutting-edge academic discovery.

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Most Cited Articles

The Most Cited Articles section features the journal's most impactful research, based on citation counts. These articles have been referenced frequently by other researchers, indicating their significant contribution to their respective fields.

• The G-Protein-Coupled Receptors in the Human Genome Form Five Main Families. Phylogenetic Analysis, Paralogon Groups, and Fingerprints

  Citation: 2374

  Authors: Robert, Malin C., Lars-Gustav, Helgi B.

• SB-431542 Is a Potent and Specific Inhibitor of Transforming Growth Factor-β Superfamily Type I Activin Receptor-Like Kinase (ALK) Receptors ALK4, ALK5, and ALK7

  Citation: 1441

  Authors: Gareth J., Francisco J., James F., John D., Laramie M., Alastair D., Nicholas J., Caroline S.

• Hypoxia-Inducible Factor-1 (HIF-1)

  Citation: 1338

  Authors: Qingdong, Max

• G Protein-Coupled Receptors as Targets for Approved Drugs: How Many Targets and How Many Drugs?

  Citation: 1079

  Authors: Krishna, Paul A.

• The Characterization of Two Specific Drug Binding Sites on Human Serum Albumin

  Citation: 1021

  Authors: G., D.J., D.N.

• Determination and characterization of a cannabinoid receptor in rat brain.

  Citation: 948

  Authors: W A, F A, M R, L S, A C

• The Nuclear Receptor Peroxisome Proliferator-Activated Receptor-α Mediates the Anti-Inflammatory Actions of Palmitoylethanolamide

  Citation: 761

  Authors: Jesse, Jin, Giuseppe, Giovanna, Roberto, Antonio, Daniele

• Sex Differences in the Expression of Hepatic Drug Metabolizing Enzymes

  Citation: 613

  Authors: David J., Minita G.

• Neuroprotection against Oxidative Stress by Estrogens: Structure-Activity Relationship

  Citation: 598

  Authors: Christian, Thomas, Frank, Anke, Martina, Christopher J., Florian

• Differential Response of Estrogen Receptor α and Estrogen Receptor β to Partial Estrogen Agonists/Antagonists

  Citation: 594

  Authors: Tomas, Bo, Yvonne, Eva, Jan-Ã…ke, Stefan

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