DMM Disease Models and Mechanisms

Aims & Scope

Disease Models & Mechanisms (DMM) publishes original research, resources and invited reviews that focus on the use of model systems to better understand, diagnose and treat human disease.

The primary aim of DMM is to promote human health by inspiring collaboration between basic and clinical researchers in translational science.

The journal is committed to presenting highly significant research that meets this goal.

The interdisciplinary nature of DMM means that a diverse range of diseases, approaches and models fall within its scope.

Examples of DMM content: -Basic research that significantly advances our understanding of human disease mechanisms, diagnosis or treatment, and is of broad relevance -Drug screens and preclinical analyses of candidate therapies using model systems -Presentation of new models that have been thoroughly validated to demonstrate their utility in disease studies* -New methods and resources that provide broadly useful tools for a disease and/or model community -Comprehensive, expert reviews and posters addressing current and emerging themes in biomedical science -Interviews with leading scientists in translational research -Editorials that discuss wider issues of interest to the community, including regulatory, ethical and policy issues -Special collections that collate articles relating to a particular disease area or model system *Descriptive work, such as the characterisation of a model system without demonstration of its usefulness to advance a field, is generally not considered for publication, unless the new model itself is considered by the Editorial team to provide an improved understanding of the disease mechanism and/or to demonstrate clear usefulness for the evaluation of treatment approaches.

Metrics & Ranking

Abstracting & Indexing

Journal is indexed in leading academic databases, ensuring global visibility and accessibility of our peer-reviewed research.

• Scopus
• Google Scholar
• Directory of Open Access Journals (DOAJ)
• Web of Science
• Medline
• PubMed
• Embase

Subjects & Keywords

Journal’s research areas, covering key disciplines and specialized sub-topics in Biochemistry, Genetics and Molecular Biology, Immunology and Microbiology, Medicine and Neuroscience, designed to support cutting-edge academic discovery.

Licensing & Copyright

This journal operates under an Open Access model. Articles are freely accessible to the public immediately upon publication. The content is licensed under a Creative Commons Attribution 4.0 International License (CC BY 4.0), allowing users to share and adapt the work with proper attribution.

Copyright remains with the author(s), and no permission is required for non-commercial use, provided the original source is cited.

Policy Links

This section provides access to essential policy documents, guidelines, and resources related to the journal’s publication and submission processes.

• Aims scope
• Homepage
• Oa statement
• Author instructions
• License terms
• Review url
• Board url
• Copyright url
• Plagiarism url
• Preservation url
• Apc url
• License

Plagiarism Policy

This journal follows a plagiarism policy. All submitted manuscripts are screened using reliable plagiarism detection software to ensure originality and academic integrity. Authors are responsible for proper citation and acknowledgment of all sources, and any form of plagiarism, including self-plagiarism, will not be tolerated.

For more details, please refer to our official: Plagiarism Policy.

APC Details

The journal’s Article Processing Charge (APC) policies support open access publishing in Biochemistry, Genetics and Molecular Biology, Immunology and Microbiology, Medicine and Neuroscience, ensuring accessibility and quality in research dissemination.

This journal requires an Article Processing Charge (APC) to support open access publishing, covering peer review, editing, and distribution. The current APC is 4,000.00 USD. Learn more.

Explore journals without APCs for alternative publishing options.

Most Cited Articles

The Most Cited Articles section features the journal's most impactful research, based on citation counts. These articles have been referenced frequently by other researchers, indicating their significant contribution to their respective fields.

• A comprehensive definition for metabolic syndrome

  Citation: 1688

  Authors: Paul L.

• Remodeling and homeostasis of the extracellular matrix: implications for fibrotic diseases and cancer

  Citation: 1338

  Authors: Thomas R., Janine T.

• How informative is the mouse for human gut microbiota research?

  Citation: 1117

  Authors: Thi Loan Anh, Sara, Adrian, Jeroen

• Cellular and molecular mechanisms of muscle atrophy

  Citation: 1021

  Authors: Paolo, Marco

• Airway basal stem cells: a perspective on their roles in epithelial homeostasis and remodeling

  Citation: 643

  Authors: Jason R., Scott H., Brigid L. M.

• Hypothalamic circuits regulating appetite and energy homeostasis: pathways to obesity

  Citation: 636

  Authors: Katharina, Jens C.

• Hooked on fat: the role of lipid synthesis in cancer metabolism and tumour development

  Citation: 630

  Authors: Franziska, Barrie, Heike, Almut

• Neurodegenerative disease: models, mechanisms, and a new hope

  Citation: 618

  Authors: Aaron D., Paraminder, James

• Standard operating procedures for describing and performing metabolic tests of glucose homeostasis in mice

  Citation: 595

  Authors: Julio E., Varman T., Gregory J., Silvana, Colleen M., Gerald I., David H., Owen P.

• Rodent models in neuroscience research: is it a rat race?

  Citation: 526

  Authors: Bart, Jiun