Experimental and Molecular Pathology
Published by Elsevier
ISSN : 0014-4800 eISSN : 1096-0945
Abbreviation : Exp. Mol. Pathol.
Aims & Scope
Under new editorial leadership, Experimental and Molecular Pathology presents original articles on disease processes in relation to structural and biochemical alterations in mammalian tissues and fluids and on the application of newer techniques of molecular biology to problems of pathology in humans and other animals.
The journal also publishes selected interpretive synthesis reviews by bench level investigators working at the "cutting edge" of contemporary research in pathology.
In addition, special thematic issues present original research reports that unravel some of Nature's most jealously guarded secrets on the pathologic basis of disease.
Research Areas include: Stem cells; Neoangiogenesis; Molecular diagnostics; Polymerase chain reaction; In situ hybridization; DNA sequencing; Cell receptors; Carcinogenesis; Pathobiology of neoplasia; Complex infectious diseases; Transplantation; Cytokines; Flow cytomeric analysis; Inflammation; Cellular injury; Immunology and hypersensitivity; Athersclerosis.
View Aims & ScopeMetrics & Ranking
Impact Factor
Year | Value |
---|---|
2025 | 3.7 |
2024 | 2.80 |
SJR (SCImago Journal Rank)
Year | Value |
---|---|
2024 | 0.803 |
Quartile
Year | Value |
---|---|
2024 | Q1 |
h-index
Year | Value |
---|---|
2024 | 81 |
Journal Rank
Year | Value |
---|---|
2024 | 6754 |
Journal Citation Indicator
Year | Value |
---|---|
2024 | 538 |
Impact Factor Trend
Abstracting & Indexing
Journal is indexed in leading academic databases, ensuring global visibility and accessibility of our peer-reviewed research.
Subjects & Keywords
Journal’s research areas, covering key disciplines and specialized sub-topics in Biochemistry, Genetics and Molecular Biology and Medicine, designed to support cutting-edge academic discovery.
Licensing & Copyright
This journal operates under an Open Access model. Articles are freely accessible to the public immediately upon publication. The content is licensed under a Creative Commons Attribution 4.0 International License (CC BY 4.0), allowing users to share and adapt the work with proper attribution.
Copyright remains with the author(s), and no permission is required for non-commercial use, provided the original source is cited.
Policy Links
This section provides access to essential policy documents, guidelines, and resources related to the journal’s publication and submission processes.
- Aims scope
- Homepage
- Oa statement
- Author instructions
- License terms
- Review url
- Board url
- Copyright url
- Plagiarism url
- Preservation url
- Apc url
- License
Plagiarism Policy
This journal follows a plagiarism policy. All submitted manuscripts are screened using reliable plagiarism detection software to ensure originality and academic integrity. Authors are responsible for proper citation and acknowledgment of all sources, and any form of plagiarism, including self-plagiarism, will not be tolerated.
For more details, please refer to our official: Plagiarism Policy.
Most Cited Articles
The Most Cited Articles section features the journal's most impactful research, based on citation counts. These articles have been referenced frequently by other researchers, indicating their significant contribution to their respective fields.
-
Discovery and development of the G-rich oligonucleotide AS1411 as a novel treatment for cancer
Citation: 730
Authors: Paula J., Damian A., Donald M., Shelia D., John O.
-
Transformation of alveolar Type 2 cells to Type 1 cells following exposure to NO2
Citation: 485
Authors: Michael J., Linda J., Robert J., Gustave
-
Origins and virulence mechanisms of uropathogenic Escherichia coli
Citation: 442
Authors: Travis J., Richard R., Matthew A.
-
Hepatic ischemia/reperfusion injury—a fresh look
Citation: 348
Authors: Constantino, Ronald W, Jerzy W
-
Regulation of glucose metabolism by 6-phosphofructo-2-kinase/fructose-2,6-bisphosphatases in cancer
Citation: 320
Authors: Abdullah, Sucheta, Brian, Jason
-
Mesenchymal stem cells derived from bone marrow favor tumor cell growth in vivo
Citation: 313
Authors: Wei, Wenrong, Runqiu, Hui, Miao, Jiabo, Weike, Chongxu, Yongchang
-
Smooth muscle phenotypic changes in arterial wall homeostasis: Implications for the pathogenesis of atherosclerosis
Citation: 301
Authors: Gordon R, Julie H