Apoptosis

Aims & Scope

Apoptosis is an international peer-reviewed journal, published monthly.

The Journal is devoted to the rapid publication of innovative basic and clinically-oriented investigations into programmed cell death.

It aims to stimulate both research on the basis of mechanisms of apoptosis and on its role in various human disease processes including: cancer, autoimmune disease, viral infection, AIDS, cardiovascular disease, neurodegenerative disorders, osteoporosis and ageing.

The Editor-In-Chief recognises the need to encourage the development of clinical therapies against apoptosis-related diseases.

Original Articles, Reviews, Short Communications, Letters to the Editor and Book Reviews will be considered for publication in Apoptosis.

View Aims & Scope

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Metrics & Ranking

Impact Factor

Year	Value
2025	8.1
2024	6.10

Journal Rank

Year	Value
2024	1170

Journal Citation Indicator

Year	Value
2024	2022

SJR (SCImago Journal Rank)

Year	Value
2024	2.233

Quartile

Year	Value
2024	Q1

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Abstracting & Indexing

Journal is indexed in leading academic databases, ensuring global visibility and accessibility of our peer-reviewed research.

• Scopus
• Google Scholar
• Web of Science
• Medline
• Embase

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Subjects & Keywords

Journal’s research areas, covering key disciplines and specialized sub-topics in Biochemistry, Genetics and Molecular Biology, Medicine and Pharmacology, Toxicology and Pharmaceutics, designed to support cutting-edge academic discovery.

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Most Cited Articles

The Most Cited Articles section features the journal's most impactful research, based on citation counts. These articles have been referenced frequently by other researchers, indicating their significant contribution to their respective fields.

• Role of reactive oxygen species (ROS) in apoptosis induction

  Citation: 2404

  Authors: H.-U., A., F.

• Mitochondria, oxidative stress and cell death

  Citation: 1618

  Authors: Martin, Vladimir, Sten, Boris

• The mitochondrial membrane potential (Δψm) in apoptosis; an update

  Citation: 1404

  Authors: J. D., D. R., A.

• PI3K-Akt pathway: Its functions and alterations in human cancer

  Citation: 989

  Authors: M., M., H.

• Apoptosis and interferons: Role of interferon-stimulated genes as mediators of apoptosis

  Citation: 691

  Authors: M., D. J., Y.-F., B. R., G. C., R. H., E. C.

• Zinc oxide nanoparticles induce oxidative DNA damage and ROS-triggered mitochondria mediated apoptosis in human liver cells (HepG2)

  Citation: 658

  Authors: Vyom, Diana, Alok

• Poly(ADP-ribose) polymerase-1 cleavage during apoptosis: An update

  Citation: 604

  Authors: C., A. I.

• Induction of reactive oxygen species: an emerging approach for cancer therapy

  Citation: 497

  Authors: Zhengzhi, Haocai, Haolong, Songmao

• Autophagy and apoptosis: where do they meet?

  Citation: 479

  Authors: Subhadip, Prashanta Kumar, Niharika, Durgesh Nandini, Sujit Kumar

• Apoptosis and autophagy: regulatory connections between two supposedly different processes

  Citation: 467

  Authors: Andrew

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