American Journal of Physiology - Lung Cellular and Molecular Physiology

Aims & Scope

The American Journal of Physiology-Lung Cellular and Molecular Physiology publishes original research covering the broad scope of molecular, cellular, and integrative aspects of normal and abnormal function of cells and components of the respiratory system.

Areas of interest include conducting airways, pulmonary circulation, lung endothelial and epithelial cells, the pleura, neuroendocrine and immunologic cells in the lung, neural cells involved in control of breathing, and cells of the diaphragm and thoracic muscles.

The processes to be covered in the Journal include gas-exchange, metabolic control at the cellular level, intracellular signaling, gene expression, genomics, macromolecules and their turnover, cell-cell and cell-matrix interactions, cell motility, secretory mechanisms, membrane function, surfactant, matrix components, mucus and lining materials, lung defenses, macrophage function, transport of salt, water and protein, development and differentiation of the respiratory system, and response to the environment.

View Aims & Scope

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Metrics & Ranking

Impact Factor

Year	Value
2025	3.5
2024	3.60

SJR (SCImago Journal Rank)

Year	Value
2024	1.240

Quartile

Year	Value
2024	Q1

Journal Rank

Year	Value
2024	3365

Journal Citation Indicator

Year	Value
2024	1750

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Abstracting & Indexing

Journal is indexed in leading academic databases, ensuring global visibility and accessibility of our peer-reviewed research.

• Scopus
• Google Scholar
• Web of Science
• Embase

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Subjects & Keywords

Journal’s research areas, covering key disciplines and specialized sub-topics in Biochemistry, Genetics and Molecular Biology and Medicine, designed to support cutting-edge academic discovery.

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Most Cited Articles

The Most Cited Articles section features the journal's most impactful research, based on citation counts. These articles have been referenced frequently by other researchers, indicating their significant contribution to their respective fields.

• Reactive oxygen species in cell signaling

  Citation: 2070

  Authors: Victor J., Barry L.

• Animal models of acute lung injury

  Citation: 1356

  Authors: Gustavo, Charles W., Thomas R.

• Unusual inflammatory and fibrogenic pulmonary responses to single-walled carbon nanotubes in mice

  Citation: 1015

  Authors: Anna A., Elena R., Robert, Ashley R., Victor J., Alla I., Yulia Y., Olga, Sevaram, Diane, Ann F., James, Douglas E., Bon-Ki, Dawn, Andrew, Valerian E., Vincent, Paul

• TGF-β-induced EMT: mechanisms and implications for fibrotic lung disease

  Citation: 831

  Authors: Brigham C., Zea

• Mechanosignaling through YAP and TAZ drives fibroblast activation and fibrosis

  Citation: 666

  Authors: Fei, David, Kyoung Moo, Lauren, Aleksandar, Vladimir, Clemens K., Samantha E., Thomas H., Jeffrey C., Ivan O., Laura E., Carol, Xaralabos, Andrew M., Daniel J.

• Murine models of pulmonary fibrosis

  Citation: 662

  Authors: Bethany B., Cory M.

• Heme oxygenase: colors of defense against cellular stress

  Citation: 657

  Authors: Leo E., Augustine M. K.

• Animal models of pulmonary arterial hypertension: the hope for etiological discovery and pharmacological cure

  Citation: 651

  Authors: Kurt R., Barbara, Nazzareno, Wolter J., Ivan F.

• NF-κB activation as a pathological mechanism of septic shock and inflammation

  Citation: 612

  Authors: Shu Fang, Asrar B.

• Epithelial repair mechanisms in the lung

  Citation: 584

  Authors: Lynn M., Christopher M.

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